Prof. André GrotenhuisNetherlands
Radboud University Nijmegen Medical Center
| 2019 to present | Emeritus professor of neurosurgery |
| 1999 - 2019 | professor, chairman & training program director |
| 1987 - 1999 | associate professor and consultant neurosurgeon |
| 2014 - 2017 | president of the EANS |
| 2020 - 2023 | president of the EANS Foundation for Research in Neurological Surgery |
| 2015 - now | president of the International Society for Neurosurgical Technology and instrument invention (ISNTii) |
Minimally Invasive Techniques, vascular neurosurgery, skull base surgery, pituitary surgery, deep-seated & intraventricular tumors
President of the International Society of Neurosurgical Technology and instrument invention (ISNTii) since 2015
Past- President of the European Association of Neurosurgical Societies (EANS)
Past- President of the EANS Foundation for Research in Neurological Surgery
Past 2nd Vice-President of the World Federation of Neurosurgical Societies (WFNS)
Elected Member of the World Academy of Neurological Surgery
Elected Member of the Academia Eurasiana Neurochirurgica
Recipient of the AANS International Lifetime Recognition Award, April 2017
Recipient of the EANS Medal of Honour, October 2022
Recipient of the Life Service Award, Croatian Neurosurgical Society, November 2022
Honorary Member of 11 Neurosurgical Societies
Knighted by the King in the highest order of chivalry (Royal Order of the Lion of The Netherlands) in 2019
The anatomical substrate of cerebellar mutism
1109 13:00-13:10
AASNS & WANS Joint Seminar/305
Cerebellar mutism is a well-known and feared complication of surgery for cerebellar tumor in childhood, although it also can occur due to other causes and also in adulthood. In former times the surgical splitting of the vermis was blamed as the cause for cerebellar mutism and neurosurgeons shifted towards the telovelar approach to avoid damaging the vermis. However, the rate of postoperative cerebellar mutism remained the same. Based upon a case of cerebellar mutism that we encountered in an adult patients with inadvertent perforator closure during removal of a prepontine epidermoid, resulting in pontine infarction in the area of the nucleus ruber and the dentate-rubro-thalamic tract led to the hypothesis that a lesion of the dentate-rubro-thalamic tract could be the anatomical substrate of cerebellar mutism. We have studied this with MRI probabalistic tractography and histoligical examinations of human brain specimen and then pre- and postoperative MRI-tractography in cases with and without cerebellar mutism. These studies confirmed that damage of the right dentate-rubrothalamic tract in left-handed patients and bilateral damage of that tract in right-handed patients, leading to a crossed cerebello-cerebral diaschisis, can be onsidered as the anatomical substrate of cerebellar mutism.